https://mgcd0103inhibitor.com/....any-missense-mutatio
In this study, we demonstrated that GM substances work as anti-TNBC representatives through activation of the JNK/AP-1 path. RNA-seq and biochemical analyses of GM compound-treated cells uncovered that c-Jun N-terminal kinase (JNK) and members of the downstream signaling pathway are possible targets for GM compounds. Mechanistically, JNK activation by GM compounds induced a rise in c-Jun phosphorylation and c-Fos protein levels, there
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