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Both loss of PSAT1 and elimination of serine through the diet had been required to suppress colorectal disease xenograft growth and enhance the antitumor activity of 5-fluorouracil (5-FU). Restricting endogenous and exogenous serine in vitro augmented 5-FU-induced cell demise, DNA damage, and metabolic perturbations, most likely bookkeeping for the observed antitumor impact. Collectively, our outcomes suggest that both endogenous and exogenous types of serine contribute to colo

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05). It was determined that the development of AKI in normal-weight and overweight patients increased mortality (   0.001) and that there was not a difference in mortality rates between obese patients with and without AKI. The risk of AKI development was higher in obese patients but not in those who were in serious conditions. Another paradox was that the development of AKI was associated with a higher mortality rate in normal-weight and overweight patients, but not in obese patients. Cerebrovascular diseases as a cause of admission