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In our design, we launched numerous moieties (catechol, non-catechol, biphenyl, piperazine, and thiazole) to determine which practical group results in the maximum aggregation inhibition of tau. In vitro, tau aggregation had been induced by heparin and monitored by making use of fluorescence aggregation assay, transmission electron microscopy and 4,4'-Dianilino-1,1'-binaphthyl-5,5'-disulfonic acid dipotassium sodium (Bis-ANS) fluorescence spectroscopy. The cate
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