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In inclusion, we investigated the role of miRs in managing endothelial nitric oxide synthase (eNOS) appearance in human being coronary artery endothelial cells (hCAECs). One of the 121 patients, 46 were diagnosed with VA (VA team), 26 with insignificant coronary lesions (ICL group), and 49 with atherothrombotic angina (AA group). The VA group revealed a significantly greater phrase of miR-17-5p, miR-92a-3p, and miR-126-3p compared to the ICL group. In contrast, miR-221-3p and