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rucial role in the progression of HCC and provides an example of how co-amplified genes work together in HCC.[This corrects the article DOI 10.7150/thno.42795.].Although dyslipidemia commonly occurs in patients with acute promyelocytic leukemia (APL) in response to anti-APL therapy, the underlying mechanism and the lipid statuses of patients with newly diagnosed APL remain to be addressed. Methods We conducted a retrospective study to investigate the lipid profiles of APL patients. PML-RARα transgenic mice and APL cells-transplanted mic
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