https://www.selleckchem.com/pr....oducts/apr-246-prima
One patient with elevated NO showed a transient neurologic deficit that resolved 3 days post-operatively. Mixed venous NO was significantly elevated shortly following cerebral artery occlusion vs. baseline in a majority of the study subjects, suggesting that NO is a potential biomarker for ischemia. Since all patients received identical burst suppression anesthesia and vasopressors, the fact that NO was not elevated during cross-clamp in all patients supports the conclusion that the NO elevation is likely due to ischemia. Mix
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