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Verification analyses yielded similar patterns of thalamic FC changes in TLE. Importantly, the decreased FC between the ANT and hippocampal pathway was correlated with the poorer memory performance of TLE. Conclusion These findings suggest that the distinct thalamocortical FC patterns are damaged to some extent in TLE patients. Importantly, the specific pathology of the ANT-hippocampal pathway in TLE may be a potential factor that contributes to memory deficits. Our study may pave the way for improved treatments and cognitive function b
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