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H S level and H S synthetase activity were decreased. Increasing the level of H S by NaHS improved the pathological changes of kidney tissues and limited the number of pyroptotic cells. In vitro, NaHS could reverse H/R-induced cell injury. H S suppressed cell pyroptosis and kidney injury via inhibiting the NLRP3/Caspase-1 axis. We highlighted that H S prevented cell pyroptosis via suppressing the NLRP3/Caspase-1 axis, thereby inhibiting I/R-induced AKI. These findings may confer novel insights for the clinical management of I/R-induced AKI. We highlighted t
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