https://www.selleckchem.com/products/rvx-208.html
MAIN RESULTS The nanoparticles efficiently targeted endothelial cells and myofibroblasts in the alveolar region. Nanoparticle delivery of either FOXM1 or FOXF1 did not protect endothelial cells from apoptosis caused by hyperoxia but increased endothelial proliferation and lung angiogenesis after the injury. FOXM1 and FOXF1 improved elastin fiber organization, decreased alveolar simplification and preserved lung function in mice reaching adulthood. CONCLUSIONS Nanoparticle delivery of FOXM1 or FOXF1 stimulates lung angiogenesis and alveo
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