https://www.selleckchem.com/pr....oducts/ly3009120.htm
Cytoplasmic and nuclear P27Kip1 showed overexpression following treatment with I3C higher than that detected following DIM treatment. This study provides a mechanistic elucidation of the previously reported cell cycle arrest by I3C and DIM in breast cancer cells suggesting that this effect could be through modulation of miRNAs expression that, in turn, regulates the genetic network controlling the G1/S phase in cell cycle progression.Enzymatic hydrolysis of naringin by the action of naringinase is one of the standard practices adopted
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