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These results seem to mirror the early deleterious aftereffects of T1D on neurodevelopment, which affects primarily interest allocation procedures and also the neurofunctional substrates that underlie all of them. Fibrinogen-like protein 2 (FGL2), a part associated with the fibrinogen superfamily, happens to be explained to augment immunosuppression in gliomas. But, the complete clinical molecular features plus the prognostic relevance of FGL2 in gliomas remain not clear. Therefore, a com
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