https://www.selleckchem.com/Proteasome.html
Using a synaptosomal preparation of rat prefrontal cortex devoid of postsynaptic elements, we found that CRF2α and D1R colocalize in synaptic terminals of the rat medial prefrontal cortex and that the simultaneous activation of both receptors also occluded phosphorylation of ERK. These results strengthen the idea that the heteromer CRF2a-D1R is an entity functionally different from each receptor that composes it and suggests that its formation is enhanced by CRF and dopamine co-transmission, as occurs in stress and addiction. This article is
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