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The pathogenesis of liver fibrosis involves liver damage, inflammation, oxidative stress, and intestinal dysfunction. Indole-3-propionic acid (IPA) has been demonstrated to have antioxidant, anti-inflammatory and anticancer activities, and a role in maintaining gut homeostasis. The current study aimed to investigate the role of IPA in carbon tetrachloride (CCl )-induced liver fibrosis and explore the underlying mechanisms. The liver fibrosis model was established in male C57BL/6 mice by intraperitoneal injection of CCl twice weekl