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Mutation of KIN10, the catalytic α-subunit of SnRK1, leads to the diminished stomatal index; while overexpression of KIN10 dramatically causes stomatal development. KIN10 shows the cell-type-specific subcellular place structure. The nuclear-localized KIN10 proteins are extremely enriched in the stomatal lineage cells to phosphorylate and support SPEECHLESS, a master regulator of stomatal development, therefore advertising stomatal development. Our work identifies a module backlinks linking th