https://www.selleckchem.com/products/LY335979.html
Clinical studies indicate that sepsis induced diaphragm dysfunction is a major contributor to respiratory failure in mechanically ventilated patients. Currently there is no drug to treat this form of diaphragm weakness. Sepsis induced muscle dysfunction is thought to be triggered by excessive mitochondrial free radical generation, we therefore hypothesized that therapies which target mitochondrial free radical production may prevent sepsis induced diaphragm weakness. The present study determined if MitoTEMPOL, a mitochondrially targete
Like
Comment
Share
