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nt diabetic cardiomyopathy. Here, we show that a complex epigenetic machinery involving DNA methylation, histone modifications, and microRNAs mediates hyperglycemia-induced JunD downregulation and myocardial dysfunction in experimental and human diabetes mellitus. Our results pave the way for tissue-specific therapeutic modulation of JunD to prevent diabetic cardiomyopathy.This paper looks at the terms and conditions of acceptance for queer and trans Muslims in dominant Muslim groups. The social tension between LGBTQ Identity and rel