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Aside from concentrating on the main inhibitory aftereffect of glial scar linked proteoglycans like chondroitin sulfate as well as the reactive glial cell released inflammatory and development inhibitory particles, the other major regeneration stalling element that occurs after extreme CNS injury is myelin and its particular associated proteinaceous debris comprising myelin-associated glycoprotein (MAG), neurite outgrowth inhibitor necessary protein (NOGO), and oligodendrocyte
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