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This choice sign is notably stronger for variations arising upstream of genes intolerant to loss-of-function variants. Furthermore, variants creating uORFs that overlap the coding sequence show signals of choice equivalent to coding missense variations. Finally, we identify particular genetics where customization of uORFs likely represents an important condition method, and report a novel uORF frameshift variant upstream of NF2 in neurofibromatosis. Our results highlight uORF-perturbing variations a
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