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We verified that the larger expression of SEC61G in HCC tissues than paracancerous cells. Moreover, knockdown of SEC61G inhibited cell expansion and induced cell apoptosis in vitro. Besides, SEC61G ended up being necessary for cell migration and intrusion, conferring a potential role for SEC61G in tumor transfer. Taken collectively, our results disclosed the part of SEC61G in HCC cells. Further detailed understanding associated with the signaling systems underlying SEC61G involvement in HCC cells would make SEC61G as a viabl
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