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Practical assays showed that FADS1 knockdown suppressed the expansion, migration and intrusion of LSCC cells, while FADS1 overexpression had the contrary results. Bioinformatic analysis centered on microarray data discovered that FADS1 could activate AKT/mTOR signaling. This hypothesis ended up being further validated by in both vivo as well as in vitro assays. Thus, our information features supported the viewpoint that FADS1 is a potential promoter in LSCC development, and has set the foundation for additional practical ana
