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has been an attractive therapeutic target in neuroblastoma considering the widespread amplification of the locus in neuroblastoma, and its established role in neuroblastoma development and progression. Thus, understanding neuroblastoma-specific control of expression at the transcriptional and post-transcriptional level would lead to identification of novel -dependent oncogenic pathways and potential therapeutic strategies. By performing loss- and gain-of-function experiments of the neuroblastoma hotspot locus 6p22.3 derived lncRNA

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