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Treatment with hesperidin also ameliorated the changed degree of inflammatory mediators and oxidative anxiety in I/R-induced renal-injured rats. The appearance of p-IκBα, caspase-3, NF-κB p65, Toll-like receptor 4 (TLR-4) protein, TLR-4 mRNA, and inducible nitric oxide synthase (iNOS) was considerably low in the renal areas of hesperidin-treated rats. Histopathological conclusions also disclosed that therapy with hesperidin attenuated the renal damage in I/R kidney-injured rats. In su
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