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Axonal transport deficit is an integral process associated with neurodegenerative conditions. Fluoxetine, a widely used antidepressant for remedy for despair, is well known to manage a handful of important architectural and neurochemical aspects of hippocampal functions. Nevertheless, the systems underlying these effects remain badly comprehended. This research aimed to research the results of persistent fluoxetine treatment on axonal transport in the hippocampus of rat stre