https://www.selleckchem.com/ph....armacological_MAPK.h
ges, with enriched pathways rearrangements in the cytoskeleton, lipid and cholesterol metabolism and upregulated genes including MVK, MSMO1, and LACC1/FAMIN. In summary, macrophages may have a central role in defining the paradigmatic cellular (T-pole) vs. humoral (L-pole) responses and it is likely that the multiplicity of infection and genetic polymorphisms in key genes are gearing this polarization. Previous research have implicated critical roles of systemic inflammation in the development of Multiple Sclerosis (MS). But the cau
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