https://www.selleckchem.com/pr....oducts/Rapamycin.htm
The role of cellular senescence in the development of asthma is not well known. We aimed to evaluate the susceptibility of mice with cellular senescence to asthma development and determine whether the mTOR pathway played an important role in this process. Cellular senescence was induced in mice by intranasal instillation of 2% cigarette smoke extract (CSE). Subsequently, a low dose (0.1 μg) of house dust mite (HDM) allergens, which causes no inflammation and airway hyperresponsiveness (AHR) in mice without cellular senescence, was adm
Like
Comment
Share
