https://www.selleckchem.com/pr....oducts/3-aminobenzam
Overall, our work supports the utility to apply personal genomic information for characterizing complex disease phenotypes.Mitochondrial dysfunction and iron (Fe) dyshomeostasis are invoked among the mechanisms contributing to muscle aging, possibly via a detrimental mitochondrial-iron feed-forward loop. We quantified the labile Fe pool, Fe isotopes, and the expression of mitochondrial Fe handling proteins in muscle biopsies obtained from young and older adults. The expression of key proteins of mitochondrial quality control (M
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