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Studies have suggested that endothelial nitric oxide synthase (eNOS) gene polymorphisms modulate the disease threat, prompting us to evaluate the influence of three functional eNOS gene polymorphisms on OSCC risk. The present study included 50 customers with OSCC and 110 controls. Polymerase sequence reaction and constraint fragment length polymorphism analysis were used for genotyping of single-nucleotide polymorphisms -786 T/C (rs2070744) and 894 G/T (rs1799983) and adju