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The SPINK1 pancreatic disease canonical path ended up being highly inhibitedf MUC1 in host protection, and offer a broad picture of alterations in cellular gene expression modulated by MUC1 in a time-dependent manner in reaction to H. pylori infection.The ADAMTS13 (a disintegrin and metalloproteinase with a thrombospondin motif repeats 13) is an integral element associated with coagulation procedure and plays an important role in the development and prognosis of persi