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Knock-down of SOX2OT led to a substantial inhibition of cell proliferation, arrested cells at G0/G1 stage and induced mobile apoptosis in MM samples in vitro, along with slowed the rise of tumours in vivo. Furthermore, our data indicated that SOX2OT functioned as a competing endogenous RNA (ceRNA) in MM cells that regulated miR-144-3p expression. Repression of miR-144-3p reversed the inhibition of MM development as a result of SOX2OT knock-down. Our information additionally disclosed tha
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