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The results revealed that an anti-67LR antibody partially abrogated the apoptosis-inducing results of EGCG on NRK-49F cells. In conclusion, EGCG may attenuate renal harm and salt-sensitive high blood pressure via exerting anti-oxidant, anti inflammatory, and apoptosis-inducing impacts on fibroblasts; the final impact is partially mediated by 67LR, suggesting that EGCG signifies a potential strategy for dealing with salt-sensitive hypertension.An amendment to the paper is publish