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In addition; we additionally observed differentially expressed genetics associated with apoptosis control, such as BCL2L1, CASP7, CDKN1A, E2F2, ITPR, CD274, TNFAIP6, TNFRSF25, TNFRSF13C, and TNFRSF17. CONCLUSION Our results claim that the mixture of altered appearance of genetics involved with signaling paths of resistant reaction and apoptosis control may add straight to the main characteristics seen in BS, such recurrent attacks, growth failure