https://www.selleckchem.com/pr....oducts/Obatoclax-Mes
Inhibiting mTOR activity with rapamycin induces raptor and restores AKT activity, allowing beta-cells to gain proliferation capacity that are lost after HFD exposure. In summary, we elucidated a novel mechanism for which lipid exposure may cause the dipole effects on beta-cell growth, where mTOR acts as a lipid sensor. These mechanisms can be novel targets for future therapeutic developments.Chronic gut inflammation such as inflammatory bowel diseases is thought as being associated with neurodegenerative diseases in humans. T
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