https://www.selleckchem.com/pr....oducts/epacadostat-i
3 channel expression in mice aorta and peritoneal macrophages which was abolished by HsTX[R14A] treatment. K 1.3 inhibition did not prevent hypertension, vascular remodelling, or stiffness but corrected AngII-induced macrophage infiltration and endothelial dysfunction in the small mesenteric arteries and/or aorta, via a mechanism independent of electrophysiological changes in VSMCs. AngII modified the electrophysiological properties of peritoneal macrophages, indicating an M1-like activated state, with enhanced expression
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