https://aurorakinaseb.com/surg....ery-management-coach
RESULTS High FKBP9 expression correlated with poor prognosis in glioma clients. Knockdown of FKBP9 markedly suppressed the malignant phenotype of GBM cells in vitro and inhibited tumefaction growth in vivo. Mechanistically, FKBP9 expression induced the activation of p38MAPK signaling via ASK1. Furthermore, ASK1-p38 signaling contributed to the FKBP9-mediated impacts on GBM cell clonogenic development. In inclusion, depletion of FKBP9 triggered the IRE1α-XBP1 pathway, which played a task in the FKBP9-m
Like
Comment
Share
