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Portal hypertension is the main initial consequence of cirrhosis and the main driver of decompensation. It follows that pathophysiological and therapeutic research studies have relied on measuring portal pressure. Because the portal vein is located between two capillary beds, the hepatic sinusoids and the splanchnic capillaries, measuring portal pressure directly is difficult. Wedging a catheter (or occluding it with a balloon) in a suprahepatic vein is less invasive and, despite being an indirect method, correlates very well with direct p