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Within our design, we introduced different moieties (catechol, non-catechol, biphenyl, piperazine, and thiazole) to determine which useful team causes the maximum aggregation inhibition of tau. In vitro, tau aggregation had been induced by heparin and monitored by utilizing fluorescence aggregation assay, transmission electron microscopy and 4,4'-Dianilino-1,1'-binaphthyl-5,5'-disulfonic acid dipotassium sodium (Bis-ANS) fluorescence spectroscopy. The catechol containing compounds, D-519 and D-520, prevent
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