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itivity. These are the first data to provide a quantitative link between prothrombotic viscoelastic coagulation profiles with the development of infection/dehiscence. Based on the cut-points of 33.2 mm MA, 46.6% platelet aggregation, or 55.8% platelet inhibition, we recommend consideration of an enhanced antimicrobial or antithrombotic approach for these high risk groups. 46.6% platelet aggregation, or less then 55.8% platelet inhibition, we recommend consideration of an enhanced antimicrobial or antithrombotic approach fo
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