https://www.selleckchem.com/products/abt-199.html
Furthermore, BCAA treatment was found to cause decreased levels of Glut-1 and phosphorylated GSK-3 and mTOR in astrocytes. Although further investigations of the effect of BCAA on human astrocytes with hyperammonemia are needed, our work demonstrates that BCAA supplementation has direct negative effects on astrocyte survival and intracellular metabolism. Although further investigations of the effect of BCAA on human astrocytes with hyperammonemia are needed, our work demonstrates that BCAA supplementation has direct negative effects on
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