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Extremely, bilateral NA (1 μg/0.2 μl) infusion to the PFC restored the recognition memory deficits in LC-lesioned rats. These findings indicate that a selective noradrenergic LC lesion induced by 6-OHDA deregulates a noradrenergic community in the PFC, that could be engaged during the early memory impairments observed in nondemented PD patients.Lung disease is the most typical and deadly malignant illness which is why the introduction of effective chemotherapeutic representatives remains
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