https://www.selleckchem.com/pr....oducts/AZD2281(Olapa
Furthermore, ER stress in KCs promoted the polarization of KCs towards the M1 phenotype and increased the expression of TNF- . The increase in KC-derived TNF- triggered the apoptosis of HSCs and the activation of TNF-R1/caspase 8 in vitro, which was consistent with the in vivo results. ER stress in KCs promotes the polarization of these cells towards the M1 phenotype and increases the expression of TNF- . Then, the increase in KC-derived TNF- triggers the apoptosis of HSCs through TNF-R1/caspase 8. ER stress in KCs promotes th
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