https://www.selleckchem.com/ph....armacological_MAPK.h
This structural abnormality was associated with the generation of higher levels of ROS and mitochondrial superoxide but lower ATP. These physiological changes in mutants did not result in discernable effects on animal motility and lifespan. Moreover, the amount of glucose in N2 wild-type worms treated with troglitazone and pioglitazone, derivatives of TZD, was reduced to a comparable level as in the mutant animals. CONCLUSIONS By focusing on the Cisd-1 gene, our study established a C. elegans genetic system suitable for modeling hum
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