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Nevertheless, the possibility healing role and components of GA in T‑cell acute lymphoblastic leukemia (T‑ALL) stay uncertain. In the present study, the effects of GA on expansion, cellular period, apoptosis, and autophagy in T‑ALL mobile lines had been examined. The possible mechanisms underlying GA task were additionally examined. The outcomes indicated that GA inhibited proliferation, induced apoptosis, and activated autophagy in T‑ALL cellular lines (Jurkat and Molt‑4 cells). Findings confirmed that GA
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