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This was validated by movement cytometry showing a G1/S stage block caused by CP655. Finally, mechanistic experiments unveiled that the chelator might be causing an upregulation of the cell cycle inhibitor necessary protein CDKN1A (p21) just as one procedure of action. In closing, this research demonstrates that HPO chelators could prove to have healing potential for conditions driven by exorbitant T cell proliferation.The finding and utilization of thiopurine methyltransferase (TPMT) pharmacogen
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