https://www.selleckchem.com/pr....oducts/salinosporami
Clone formation showed that elevated BTG2 promoted DNA damage of irradiated H460 cells. The number of γ-H2AX foci induced by DNA damage was also markedly increased upon BTG2 overexpression. Flow cytometry showed that BTG2 increased IR-induced cell apoptosis. BTG2 may be a novel radio-responsive factor and a promising therapeutic target for radiotherapy of NSCLC. BTG2 may be a novel radio-responsive factor and a promising therapeutic target for radiotherapy of NSCLC.Assistive eco-physiological traits are nece
Like
Comment
Share
