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This review centered on the components of folate k-calorie burning and related substances affecting S. aureus disease. Lack of tetrahydrofolic acid (THF)-dependent dTMP directly inhibits the nucleotide synthesis path associated with the S. aureus due to pabA deficiency. Besides, trimethoprim-sulfamethoxazole (TMP/SMX), a potent antibiotic drug that treats S. aureus infections, interferes along the way for the folate process and contributes to the production of thymid
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