https://www.selleckchem.com/
The results indicate a new possible therapeutic strategy against multiple resistant A. baumannii. Research has advanced our understanding of the molecular and cellular mechanisms of cerebral edema and has propelled the development of novel antiedema therapeutics. Current evidence supports aberrant neuro-glial ion transport as a central mechanism that underlies pathological fluid accumulation after central nervous system injury. Novel agents in clinical development show potential in altering the natural history and treatment of cerebral edema. Using the PubM
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