https://www.selleckchem.com/products/ho-3867.html
S1P deficiency affects both the trafficking of activated peritoneal B cells to secondary lymphoid organs and the positioning of these cells within the functional compartments of the targeted organ. S1P deficiency in LPS-activated peritoneal B cells results in significantly reduced numbers of splenic innate response activator B cells. The S1P-S1PR system is implicated in the trafficking of LPS-activated peritoneal B cells. Given the protective role of peritoneal B1a B cells in peritoneal sepsis, further experiments to investigate the imp
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