https://www.selleckchem.com/products/gne-049.html
034) and SW (all p less then 0.04, and non-target early P3a (eP3a; all p less then 0.023) and late P3a (lP3a; all p less then 0.035) amplitudes were smaller for the endometriosis compared to the healthy control group. Lower non-target eP3a (p less then 0.001), lP3a (p = 0.013), and SW (p = 0.019) amplitudes were correlated with higher pain severity scores. Findings suggest that endometriosis-associated chronic pelvic pain is linked to alterations in stimulus-response processing and inhibitory control networks, but not impaired behavio
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