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The CX3CL1/CX3CR1 axis mediates recruitment and extravasation of CX3CR1-expressing subsets of leukocytes and plays a pivotal part into the inflammation-driven pathology of heart disease. The cardiac immune response varies with respect to the fundamental causes. This suggests that when it comes to growth of effective immunomodulatory treatment in heart failure because of persistent pressure overload induced left ventricular (LV) hypertrophy, the root protected habits should be examined. Righ