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g. KRT6A, KRT5) and negative regulators of breast tumorigenesis (e.g. EMP1). Gene methylation status was analyzed to determine whether the changes in expression induced by vitamin D compounds occurred via this mechanism. Ingenuity pathway analysis was performed to identify upstream regulators and downstream signaling pathway genes differentially regulated by vitamin D, including TP63 and vitamin D receptor (VDR) mediated canonical pathways in particular. This study provides a global profiling of changes in the gene signature of DCIS regula
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